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While same mitochondrial problems are present in the UMNs of ALS patients with TDP-43 pathology and UMNs of TDP-43 mouse models, and since pathologies are shared at a cellular level, regardless of species, we first analyzed the metabolite profile of both healthy and diseased motor cortex to investigate whether metabolomic changes occur with respect to TDP-43 pathology

Additionally, in a mouse model of PD characterized by alpha-synuclein (-syn) accumulation, BAMs, rather than microglia, were identified as the key antigen-presenting cells responsible for mediating CD4 T cell-driven neuroinflammation [179]

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2010, September: Jacksonville Mobility Summit, presenter on Regionalization and co-presenter with Congressman John Mica