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Kaibuchi, K., Takai, Y

Homocysteine may also react with nitric oxide in the body to form S-nitrosohomocysteine, a compound that causes endothelial damage and reduces the vasodilatory function of blood vessels.2,21 The primary goal in treating hyperhomocysteinemia is to reduce elevated plasma homocysteine levels in order to minimize the risk of neurological and vascular complications

Expanded NK cells were cultured in RPMI (Corning) supplemented with 10% FBS (Genesee Scientific) + 1% of each of the following: penicillin/streptomycin (HyClone), NEAA (Lonza), L-glutamine (Sigma), sodium pyruvate (Lonza), and HEPES (ThermoFisher)