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However, the central effects of chemotherapy neurotoxicity are mainly related to cognitive impairment (chemofog or chemobrain) of the central nervous system in OIPN [88, 131, 132]

Mechanistically, VDR activation acts as a potent antagonist to NF-B signaling, effectively suppressing the transcription of pro-inflammatory cytokines such as TNF-, IL-6, and IL-1

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In vitro studies showed that PROTAC 61 (LWY713) exhibited nanomolar degradation potency (DC 50 = 0.64 nM, D max = 95%) in FLT3-ITD AML MV4-11 cells