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Conclusion: This whole-exome analysis provides insights into the genetic characteristics of MVI in HCC patients, contributing to the understanding of the underlying molecular and physiological mechanisms and serving as a basis for targeted treatments for MVI

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The problem is when the body cannot recycle it efficiently

Vitamin B6 in its less common form of pyridoxal 5-phosphate (PLP) may be less likely to cause neuropathy at high doses

SLC7A11, which is not present in naive human CD4+ T cells, is substantially increased due to the activation of T cells (85, 86)