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74 The enhanced oxidant stress in the mitochondrial matrix is then mainly derived from complex I and, to a lesser degree, from complex III, 71 and the oxidant stress and injury correlate with protein adduct formation in mitochondria

APAP highly induces the expression of TGF-1 and its downstream signaling in hepatocyte necrotic regions, and blocking TGF-1 signaling by TGF receptor 1 inhibitor (GW788388) ameliorates hepatocyte inflammation and hepatocyte injury [245]

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