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Conclusions HT is an autoimmune disease with multifactorial and still not entirely known etiology

These involve substantial volume loss, dermal thinning, and sometimes underlying fat pad changes that topical treatments alone cannot fully address

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Clinically, placental hypoxia in PE induces abnormal secretion of HIF-1, which promotes the release of antiangiogenic proteins such as sFlt-1, leading to endothelial dysfunction and reduced vascular endothelial growth factor (VEGF) expression [5]

f , g , Individual tumors (left) and tumor weight (right) of PDX models (CO-04-0070 ( f ) and CO-04-0108 ( g )) treated with different combinatorial regimens