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According to the working model, these cysteine residues may undergo chemical modification/remodeling including trisulfide bond formation and/or related reactions, resulting in a regional (minor, but effective) structural alteration that enhances c-met binding and the nitration resistance of Y198-HGF

180 Cu complexes in cancer therapy The altered regulation of Cu homeostasis in tumors, coupled with the important role that Cu may play in promoting the progression of cancer, has led to the development of Cu-coordinating compounds for use in anticancer therapies

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This dual mechanism of action promotes enhanced intestinal mucosal repair