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Improved understanding of modes of action and mechanisms of resistance has resulted in rationally designed CD38-based combination therapies, which will contribute to further improvement in outcome of MM patients

The mechanism involves upregulation of VEGF expression a key growth factor in new blood vessel formation and activation of ERK1/2 signaling pathways that drive cell proliferation, migration, and angiogenesis

3.6 Anti-CD38/ICI combination therapy is ineffective in a variety of cancer types To assess the efficacy of combining anti-CD38 and PD1/PDL1-directed ICI in more generalizable scenario, we did a comprehensive literature search and compared the objective response rate (ORR) of small scale- and clinical trial-studies that administer either a combination of anti-CD38/ICI or ICI alone in various forms of cancer ( Table 2 )

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Plants are limited by vegetative reproduction and consequently rely upon a variety of dispersal vectors to transport their propagules, including both abiotic and biotic vectors