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pylori infection is a known carcinogen and a strong risk factor for non-cardia GC by the classical histopathologic Correa cascade, consequently resulting in GC ( Helicobacter pylori infection is particularly associated with an increased risk of not only non-cardia GC but also cardia GC according to several studies targeting East Asian countries, such as Korea ( Dietary effects have been reported to mediate the risk of cancer by playing a role in either the prevention of cellular carcinoma or diet-induced carcinogenesis ( Helicobacter pylori infection is required to consider the causes of GC derived from either direct or indirect inflammation in the gastric mucosa ( H

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High plasma levels were effectively associated with increased plasma and lymphocyte SAH and a decreased SAM to SAH ratio that could negatively affect cellular methylation potential (Fu et al, 2000

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