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Description

Glucose deprivation induces heme oxygenase-1 gene expression by a pathway independent of the unfolded protein response

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Seveljevic-Jaran et al., 2006

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The version listed here is the No DAC form of CJC-1295, meaning it is supplied without the Drug Affinity Complex modification that extends the half-life of CJC-1295 DAC

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Most evidently, ER stress is induced by deletion of ER chaperones, overexpression of constitutively misfolded proteins, and disruption of N-linked glycosylation, disulfide bond catalysis, or calcium homeostasis

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4 p = 0.0288, *

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