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This compound's most distinctive proposed mechanism involves its ability to bypass conventional receptor-mediated pathways and directly influence nuclear processes: Cellular and Nuclear Membrane Penetration : Due to its minimal molecular size and structure, this compound is hypothesized to passively diffuse across lipid bilayers, traversing both cellular and nuclear membranes without requiring active transport mechanisms [1] [2]

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As a natural pharmacological agent, NSO or TQ show protection against CP-induced gastrointestinal damage

Dargan Department of Environmental Health, Arak University of Medical Sciences, Arak, Iran Reza Darvishi Cheshmeh Soltani School of Public Health, University of Technology Sydney, Sydney, New South Wales, Australia Daniel Demant School of Public Health and Social Work, Queensland University of Technology, Brisbane, Queensland, Australia Daniel Demant Department of Forensic Medicine, University of Sarajevo, Sarajevo, Bosnia and Herzegovina Emina Dervievi Clinical and Public Health Research, Independent Clinician Scientist and Public Health Researcher, Ahmedabad, India Hardik Dineshbhai Desai Public Health and Environment Research Centre (PERC), Lalitpur, Nepal Keshab Deuba Global Health Epidemiology Research Group, University of Bergen, Bergen, Norway Keshab Deuba Research and Development Cell, Dr