The pathological features of CAG mainly include gastric mucosal thinning, gastric mucosal gland atrophy, elevated gastric juice pH, and decreased pepsin activity (PA) [2,3]
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Our earlier studies demonstrated that p-AMPK drives MSCs to become osteocytes, whereas under diabetes conditions, the loss of AMPK activity correlates with ubiquitination of RUNX2 and favors adipogenesis
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Nowadays, although more than one dozen of new anti-epileptic drugs have been introduced in the past 20 years, approximately one-third of patients fail to achieve complete remission (Wang and Chen, 2019)
