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Thus, a partial cure is considered an intermediate endpoint, and a functional cure continues to be the preferred endpoint of novel therapies in the development for CHB [12]

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Conversely, clinical studies have demonstrated that GLS1 promotes mitochondrial respiration rate and tumour growth potential [17]

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11,12 However, the inhibition of the xCt cystine/glutamate antiporter failed to trigger ferroptosis, despite its recognized role as a key regulator of this process