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In addition, the proinflammatory markers, such as CD16, CCL3, IL-1 and TNF- increased in Sirt3 f/f Cx3cr1-Cre mice compared with Sirt3 f/f mice at day 3 after ICH, while the anti-inflammatory markers decreased at day7, suggesting that IF may act through microglial Sirt3 to manipulate neuroinflammation after ICH

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It appears to stimulate the expression of growth factors and support the structural repair of axons and myelin sheaths, highlighting its potential as a therapeutic agent for various forms of neuropathy and nerve damage

I guess they controlled for age somewhere, but I find it hard to believe that a 30 year old with 120 IGF-1 has a lower ACM than a more age appropriate number like 180-200 (off the top of my head), simply because 120 would be indicative of an underlying health issue

Sangalli-Leite et al., 2011