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Besides the increase in melanin content, AL lesions are characterized by epidermal alterations such as proliferation/differentiation disbalance or mitochondrial dysfunction, dermal photoaging marks, and disorganization of the dermal extracellular matrix (ECM) [21,22,23]. In particular, AL lesions present an impairment of the cutaneous structure with a marked elongation of the dermalepidermal junction (DEJ), an accumulation of melanin in the epidermis, especially in the rete ridges [21]

Its often mistaken for other conditions, like eczema, contact dermatitis, or others, says Dr

doi: 10.1007/s12035-017-0695-z 45 MaggeRSDeAngelisLM

Why is understanding the AOD-9604 half life important for research

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