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10.1038/s41467-020-19579-y 20 MizumuraK.TakedaK.HashimotoS.HorieT.IchijoH

It has been reported that EGLN2 (Egl-9 family hypoxia inducible factor 2) is a candidate driver of iron chelation-mediated inhibition of cell death, and the hydroxylase activity of EGLNs is iron-dependent[114, 115]

Kaposis sarcoma-associated herpesvirus latent gene vFLIP inhibits viral lytic replication through NF-kappaB-mediated suppression of the AP-1 pathway: a novel mechanism of virus control of latency