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Baseline characteristics of the patients grouped by survival status at one year were summarized and compared

This finding provides compelling evidence that GGCT reduces ROS primarily by regulating Gln shunting to GSH, rather than through modulating energy metabolism pathways to exert its pro-proliferative effects

This suggests that GHRP-6 not only counteracted Dox-induced pro-apoptotic signaling but also enhanced protective survival pathways in cardiomyocytes [1]