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In vitro experiments using a Bcl-2 inhibitor and adeno-associated virus (AAV)-mediated Bcl-2 knockdown confirmed that the protective effects of MOTS-c against NASH-induced mitochondrial dysfunction, inflammation, and fibrosis are dependent on Bcl-2 function

Prime examples of such regulation are found in embryonic development of all multicellular organisms

Consequently, leveraging the inherent biological properties of NPPCs represents a promising therapeutic opportunity

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