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For long, it has been known that TNF- is the most prominent among them [34, 35], and some authors have reported that TNF- is able to induce muscle protein loss [36, 37] and skeletal myocyte apoptosis both in vivo and in vitro [38, 39]

The cell suspension was plated in 24 well plates and confluent cultures were treated with DMEM/F12: Trypsin (0.25%) in 1:1 ratio for 3045 min

In the hapten model and the pro-hapten model, the drug or a metabolite, respectively, covalently modifies the presented peptide, making a previously nonimmunogenic peptide immunogenic

[Google Scholar] Perry CM (2013)

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