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Compromised intestinal epithelial barrier due to intestinal epithelial-specific loss of JAM-A induces adaptive immune compensation that protects from colitis The findings in mice manipulated for MLCK or occludin expression differed from those obtained in mice lacking the JAM-A (encoded by F11r)

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Immunomodulatory biomaterials for implant-associated infections: from conventional to advanced therapeutic strategies

Research Priorities and the Translational Opportunity Despite limited clinical evidence, the biologic plausibility and widespread exposure base represent a translational opportunity for orthopaedics