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This complex symptomatology reflects the widespread and multisystem nature of PD pathology, which extends far beyond the basal ganglia to involve cortical, limbic, and peripheral autonomic structures [3, 4]

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Similarly, the minimum abundance of Akkermansia or Bifidobacterium needed to exert antidepressant effects has not been defined, which limits the development of targeted microbiota interventions

Reduced connectivity in the prefrontal cortex and anterior cingulate gyrus and reduced neuroplasticity are found in depressive disorders and play important roles in the pathophysiology of depression (66)