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In vitro, Se/LDH@PEG-M effectively inhibits pro-inflammatory M1 polarization while promoting anti-inflammatory M2 phenotypes by suppressing multiple inflammatory pathways, restoring redox homeostasis, and enhancing mitochondrial oxidative phosphorylation and ATP production via respiratory chain activation

Three different L-sites were described in GSTs from plants (Figure 1)

The hematologic and gastrointestinal abnormalities in cobalamin deficiency are similar to those of isolated folate deficiency and are generally attributable to impairment of DNA synthesis in the rapidly dividing cells of the gastrointestinal tract and bone marrow

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Ethics statement The animal study was reviewed and approved by Kyoto University Animal Research Committee