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18 Topically applied recombinant human TRX-1 suppressed the local inflammatory response evidenced by a reduction of cytokines and chemokines, such as TNF-, IL-1, IL-6, CXCL1, and MCP-1 in the dermal tissue

However, the use of these is limited by poor supplement bioavailability and several side effects [33, 34]

Similarly, redox-responsive carriers incorporating disulfide bridges (cleaved by elevated glutathione in activated microglia or neurons) liberate bacosides or quercetin exactly at sites of neuroinflammation, amplifying Nrf2 activation and neuroprotection while remaining inert during circulation (180)