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Available evidence suggests that the occurrence of HACI involves multiple interacting pathological processes, in which aggravated oxidative stress and neuroinflammation have been repeatedly reported and are considered to be one of the key mechanisms involved (Shushanyan et al., 2024

These include anemia, neurological issues, and a higher risk of heart disease

Although some conservative treatments, such as rest, nonsteroidal anti-inflammatory medication, and physical therapy, can significantly alleviate symptoms of TD, there remains a shortage of therapies that can modify the underlying pathology of the condition [22]

Colegio OR, Chu NQ, Szabo AL, et al

However, roughly 25% of people who survive a ruptured brain aneurysm have cognitive deficits or discrete neurologic deficits like difficulty speaking, weakness, and other symptoms stemming from permanent brain damage