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Banerjee et al., 2021)

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Researchers have demonstrated that amino acid metabolism is related to ferroptosis and that CCH can cause neuronal depolarization to release excess glutamate during the pathogenesis of VD, resulting in excitotoxicity, and high levels of glutamate inhibit the function of System Xc-

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Supporting the translational relevance of our findings, analysis of publicly available transcriptomic datasets (GEO: GSM8328845 and GSM5557972) revealed a consistent trend toward increased MLKL expression in human AAA tissue (Supplementary Fig

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