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statins) which have demonstrated in vitro inhibition of neutrophil migration and epithelial cell production of chemoattractants and proteases and potentiation of macrophage efferocytosis.51,52 As efferocytosis appears to be involved in the pathogenesis of a variety of chronic lung and systemic inflammatory disease including BE, this would seem an attractive target for statins therapy.53 There are currently ongoing clinical trials to evaluate the effect of atorvastatin on patients with BE, with and without P

The expression of these genes is highly linked with the development of drug resistance in lung cancer

M1, macrophage M1

COA documentation for each compound separately remains essential regardless of the experimental design