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Cordycepin prevents radiation ulcer by inhibiting cell senescence via NRF2 and AMPK in rodents

About this article Cite this article Das, S., Ahmad, Z., Singh, S

In the absence of Sox2, acinar but not ductal cells exited the cell cycle, as shown by the decrease in cyclin D1 (CCND1) + acinar cells (Fig EV2D

SS-31 may also inhibit the opening of a channel in the inner membrane called the mitochondrial permeability transition pore (mPTP), at least in rodents.5 Because mPTP opening has been implicated in neuronal death, SS-31s inhibitory action is a potential mechanism of action for the peptides cognitive health benefits.6 Furthermore, several studies have concluded that SS-31 is capable of scavenging mitochondrial reactive oxygen species, making it a potent antioxidant.19 Its antioxidant mechanism of action isnt yet fully understood, but one group of researchers has theorized that it has to do with an ability to activate autophagy20 a cellular recycling process that promotes efficient function and which is critical for mitigating oxidative stress.21 Current therapeutic uses of SS-31 Clinical studies on SS-31 support its use as a treatment for multiple aging-related changes, including those related to cognition, physiology, and vision

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