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In a mouse PD model induced by -synuclein preformed fibrils (PFFs), EGCG treatment decreased TNF- and IL-6 expression, reduced pathological aggregation, increased survival of dopaminergic neurons in the substantia nigra, and improved both motor and anxiety-like behaviors (53)

Rhee CK, Chau NQ, Yunus F, Matsunaga K, Perng DW, on behalf the CAotA

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(55) further demonstrated that in models of PD and other neurodegenerative diseases, EGCG exerts neuroprotective effects via NF-B signaling inhibition by blocking IB phosphorylation and suppressing p65 nuclear translocation, thus downregulating pro-inflammatory cytokines such as TNF- and IL-1 and reducing dopaminergic neuronal loss and motor dysfunction

The pro-inflammatory cytokines also play a crucial role in the neurodegenerative method, contributing to neuronal injury and cell death (35, 36)