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It has been well-described that DMF activates the Nrf2 signaling pathway through the electrophilic modification of Kelch-like ECH-associated protein 1 (35)

Inhibition of IDO by administering fludarabine and cyclophosphamide can restore the activity of CD19-specific CAR-T cells against IDO-positive lymphoma in vivo , suggesting that IDO is a potential therapeutic target for enhancing the clinical activity of CAR-T therapy (150, 151) ( Figure 1 )