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There is promising increased activity in the field for the development of neuroprotective and remyelinating therapies including mechanisms to support mitochondrial function and cell-based therapies targeting culprits of chronic inflammation

NijhofISLammertsvan Bueren JJvanKessel BAndrePMorelYLokhorstHMet al

Webster (Heartlands Hospital pathology department, Birmingham)

Mitochondrial function and morphology are impaired in parkin-mutant fibroblasts