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Some evidence suggests that in AD, dysregulation of tight junction proteins, increased matrix metalloproteinase signaling, and degeneration and loss of pericytes may all contribute to BBB leakage, leading to the accumulation of numerous blood-derived neurotoxic proteins in the brain, causing neuroinflammation and oxidative stress

Santos KG, Canani LH, Gross JL, Tschiedel B, Souto KE, Roisenberg I

Mitochondria serve as the primary energy-generating organelles within cells and are well-established targets of toxicity induced by various drugs and xenobiotics [42]

H 2 O 2 is the first stable compound produced in the cells as a result of stress 49

Conversely, VRK2-low PC cells exhibit impaired SLC7A11 membrane localization and intrinsic oxidative stress, creating a therapeutic window for GSH inhibition