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Molecular mechanisms regulating NPPCs in IDD Under conditions of elevated oxidative stress, excessive reactive oxygen species (ROS) accumulation and mitochondrial damage activate senescence pathways like P53/P21, contributing to accelerated ECM catabolism and NPPC senescence [76]

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One commenter stated that we should determine what level of APCM a beneficiary qualifies for to reduce practitioner burden

and exposure to ticks or areas where Lyme disease is endemic