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In this study (8) it was suggested that different IGF-1 analogs, including IGF-1 LR3, have the potential to counteract the negative action of myostatin protein, possibly helping to prevent apoptosis and protecting muscle cells

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NADPH oxidases (NOX/DUOX) For NADPH oxidases (NOX/DUOX), the rationale is to curb pathological microdomain activityfrom NOX1/2/4/5 and DUOXthat drives endothelial dysfunction, fibrosis and innate immune priming, while preserving the tightly tuned H 2 O 2 pulses required for physiological signalling, hence selective attenuation (by isoform and locale) is preferred over global blockade (Altenhfer et al