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Much of our understanding of the pathobiology of TBI has arisen from animal models that mimic features of human TBI

1-alpha, 25-dihydroxy vitamin D3 inhibits osteoclastogenesis through ifn-beta-dependent nfatc1 suppression

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Consequently, Cacna1c knockdown may act as a form of preconditioning that prevents glutamate toxicity via complex IV inhibition and concurrent decrease in mitochondrial ROS production 30