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Cagnin, Gerhard, and Banati [79] briefly outline the rationale for employing PET for imaging of activated microglia to detect in vivo inflammatory changes occurring in a variety of brain diseases and at different disease stages, and to monitor the progression of neuroinflammation as an in vivo marker of disease activity

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They suggested that acquired cysteines often play a role in tumourigenesis [113], probably by contributing to tumour suppressor genes inactivation and oncogenes activation

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Severe AMS, however, is associated with the development of other high-altitude illnesses, such as high-altitude cerebral edema (HACE) and/or high-altitude pulmonary edema (HAPE), both potentially life-threatening and characterized by vasogenic edema, altered vascular permeability, and neurological or respiratory dysfunction (Moraga et al., 2007