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Activated AKT1 phosphorylates PRMT7 at T73 residue thereby increasing the GLUD1 stabilization through PRMT7-mediated methylation and inhibiting GLUD1 ubiquitin-dependent degradation, which ultimately facilitates gastric cancer cell proliferation and migration

Although TfR-mediated transcytosis and related strategies can improve CNS delivery, clinical deploymentparticularly in the setting of anti-angiogenic-therapy-induced BBB remodelingmust balance molecular size (tissue penetration), half-life, and effector function

However, its potential role in cancer treatment is still unclear

Unlike other peptides like TB-500, BPC-157 has a unique origin and mechanism of action that targets connective tissue repair directly

MET15 / met15 0