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doi: 10.1158/2159-8290.CD-20-0844 Summary Keywords cancer, Krebs cycle, mitochondria, Complex I, heart, liver, kinetic modeling, metabolic control analysis Citation Hernndez-Esquivel L, Del Mazo-Monsalvo I, Pacheco-Velzquez SC, Feregrino-Mondragn RD, Robledo-Cadena DX, Snchez-Thomas R, Jasso-Chvez R, Saavedra E and Marn-Hernndez (2025) Modeling Krebs cycle from liver, heart and hepatoma mitochondria, supported Complex I as target for specific inhibition of cancer cell proliferation

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Consequently, the absence of GLRX leads to enhanced PSSG in response to diverse pro-inflammatory mediators, growth factors, or metabolic disease conditions 6,7,8,9,10

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