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1999 Implication of mitochondria-derived reactive oxygen species, cytochrome C and caspase-3 in N-(4-hydroxyphenyl)retinamide-induced apoptosis in cervical carcinoma cells Oncogene 18 : 63806387 Sanchez A, Alvarez AM, Benito M, Fabregat I

Additionally, their presence modulates epithelial responses to cigarette smoke by influencing cytokine signaling, oxidative stress adaptation, and tissue repair mechanisms, providing a more physiologically relevant model for inhalation toxicity studies 78,79,80