However, the limitations of neurotransmitter-based medications and the focus on other hypotheses appear to hinder further investigation into the mechanisms of excitotoxicity
In each case, the homozygous-mutant zygote proliferates to yield thousands of TrxR1-deficient cells prior to embryonic failure
thaliana leaves when inoculated on leaf surfaces (Kawaguchi et al., 2011), indicating that methanol-utilizing ability is essential for leaf colonization
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This suggests that the pathological processes and tissue damage established over years of CFTR dysfunction may not be entirely reversible by the current levels of CFTR correction achieved, or that the restoration of CFTR function is insufficient to fully extinguish these self-perpetuating cycles of inflammation and oxidative damage
