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demonstrated that suppressing the expression of SLC31A1, a key mediator of cellular Cu uptake in eukaryotic cells [Citation10], rescued renal function, improved mitochondrial activity, and inhibited apoptosis in AKI mice [Citation11]

FIGURE 6 Cellular Validation of the Specificity of GPX4 Activator To further evaluate the selectivity and specificity of compound 102 in a cellular context, we employed the cellular thermal shift assay (CETSA

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NHPN-1010 promoted functional normalization from noise-induced hearing loss as reflected in electrophysiological measures With pre-exposure measurements serving as the baseline, auditory sensitivity was evaluated by ABR recordings following noise exposure and subsequent treatment in the chronic phase