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A complex network of the m6A-metabolite-TME axis has been proposed, i.e., the ability of m6A to induce aberrant changes in metabolite levels, which may in turn trigger oncogenic signaling pathways leading to significant alterations within the TME [43]

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Erythrocyte and plasma amino acids Levels of free GLY were found to differ between the test and control diet groups in the GLY supplementation study (Fig