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This review discusses metabolic signaling pathways pertaining to cysteine disposition in the brain during normal and pathological conditions highlighting recent findings on cysteine metabolism during aging and in neurodegenerative conditions such as Huntingtons disease and molybdenum cofactor deficiency among others

To examine the nitric oxide-dependency in this system, the rings were pretreated with 0.03 mM L-NAME, an eNOS inhibitor or 10 M hemoglobin, a nitric oxide scavenger

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