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These results indicated a dynamic polarization plasticity during inflammatory progression, as evidenced by the transition from M1-dominance in acute infection to M2-preference in chronic phases ( In vivo , chronic pulmonary inflammation resulting from prolonged exposure to CS can induce the highly expressed CD206 and TGF- in macrophages, driving the adaptive immune response of the M2 phenotype to participate in tissue repair ( In vivo and in vitro , M2-directed polarization related to the TGF-/Smad pathway in COPD was observed ( As illustrated in Figure 1 , AMs play an important role in the pathogenesis of COPD by driving chronic inflammation and tissue destruction

10.1007/s10549-011-1604-1 Breast Cancer Res