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That said interest does not equal proof

13 (2), e1005284 (2017)

This IGF-1 independence eliminates a key confounding variable, allowing clean investigation of lipolytic pathway activity in isolation

10.1128/aem.01553-23 [DOI] [PMC free article] [PubMed] [Google Scholar] Hirao R., Takeuchi H., Kawada J., Ishida N

How This Works IGF-1 LR3 is a synthetic analog of human insulin-like growth factor-1 engineered with an N-terminal extension (13 amino acids) and a glutamic acid substitution at position 3, resulting in significantly reduced binding affinity to IGF binding proteins [1]