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Conversely, after APAP injection in hepatic specific PGC-1 overexpressing mice, the high expression of primary ROS scavengers is not able to counteract the hepatic oxidative stress induced by APAP, due to the collapse of GSH reservoir caused by the lowered Gcl-c expression and by the massive utilization of GSH for NAPQI detoxification performed by Glutathione S-Transferases

doi: 10.1016/j.bbi.2010.04.012 230 KuratsuneH

FIGURE 6 A correlation analysis was conducted to investigate the relationships between gut microbiota and indicators of inflammatory injury and oxidative stress induced by DSS