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Methods In order to gain insights into the molecular pathways underlying progression of emphysema and explore computational strategies for identifying COPD therapeutics, we profiled gene expression in lung tissue samples obtained from regions within the same lung with varying amounts of emphysematous destruction from smokers with COPD (8 regions 8 lungs = 64 samples)

This classification is similar to most countries worldwide, where BPC-157 remains in research chemical status pending completion of human clinical trials