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BET bromodomain inhibition rescues PD-1-mediated T-cell exhaustion in acute myeloid leukemia

Although more studies are needed to further elucidate the processes involved, it has been shown that oxidative damage of cardiomyocytes and DNA leads to cell dysfunction and apoptosis which in turn leads to electrical instability and prolonged refractory periods seen clinically on the ECG as conduction disease (49, 57)

In the case of 43 , a water-mediated hydrogen-bond to the side chain of Ser92 ( Figure 5A )

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10.1089/ars.2013.5401 206 WuJ