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Interestingly, the use of other metabolic substrates, such as pyruvate or acetate, did not protect OGD-exposed neurons 53 suggesting that lactate-mediated post-ischemic neuroprotection cannot be solely attributed to its proposed role as a metabolic fuel for neurons, but may also involves other unknown complementary mechanisms

any of the reducing agents can be prepared as a pharmaceutically acceptable salt

doi: 10.3390/ijms23052687 291

In sepsis, excessive neutrophil activation and NET release shift the endothelium from an anti-inflammatory and anticoagulant phenotype to a pro-inflammatory and pro-coagulant phenotype

Batch-specific documentation should be consulted directly when available