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found that selenium deficiency in RAW 264.7 macrophages increases oxidative stress and enhances iNOS expression and NO production through heightened NF-B activation, suggesting seleniums key role in regulating inflammatory responses (Prabhu et al., 2002)

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We thus speculate that the decline in 4-HNE load could be of importance for specific maturation checkpoints during OL differentiation and enables more efficient intracellular processes generally