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Droog et al., 1995)

Additionally, exposure of erythrocytes to heavy metals, pollutants, and xenobiotics triggers eryptosis through various mechanisms of cell death

Catalytic promiscuity and heme-dependent redox regulation of H2S synthesis

Genetic impairments in folate enzymes increase dependence on dietary choline for phosphatidylcholine production at the expense of betaine synthesis

Disturbances in the endoplasmic reticulum can be triggered by increased oxidative stress, deposition of saturated fatty acids, hypoxia, or other stimuli, which in turn cause the accumulation of unfolded proteins in the endoplasmic reticulum lumen that is filled with proteins, antioxidants, or essential metals